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Ferroptosis in Parkinson's disease: glia–neuron crosstalk: Trends in Molecular Medicine
Activation of NF-kappa B by nontypeable Hemophilus influenzae is mediated by toll-like receptor 2-TAK1-dependent NIK-IKK alpha /beta-I kappa B alpha and MKK3/6-p38 MAP kinase signaling pathways in epithelial cells
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Second, T cell senescence decreases their proliferation, cytokine production, and cytotoxicity and increases the expression of inhibitory receptors and pro-apoptotic molecules (264, 265)
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