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While the target of anticonvulsants is not specific to pain pathways, blocking ion channels on the trigeminal nerve, in theory, would therefore decrease its electrical activity and diminish CGRP release
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عنوان:العنوان الافتراضي
The differential diagnosis includes transient G6PD deficiency, as seen in typhoid fever (239), and acquired G6PD deficiency, described in a patient with acute myeloid leukemia (240)
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