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In this case, within the stellate follicle cell population of the adenohypophysis there is a subpopulation of pituitary dendritic cells, which play a role in communicating immune activation to the HPA axis and express HLA-II, and may be a target of EBV infection in some patients (56, 57)
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Other factors contributing to ASD-related neuroinflammation include microglial activation, autoimmune reaction, and genetic predisposition [58]
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