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[8] Inborn-error and cell-complementation studies Studies of rare monogenic disorders — using patient fibroblasts, complementation grouping, and enzymatic assays — provide the most direct causal evidence that a specific gene defect changes cobalamin handling and form responsiveness

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Common knowledge holds that coronavirus Spike proteins control binding to the ACE2 receptor and subsequent fusion of the viral and the plasma - or endosomal membrane - of host cells, to facilitate viral entry (186, 270) ( Figure 3 )