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Heterologous expression of γ-glutamyl transpeptidase from Bacillus atrophaeus GS-16 and its application in the synthesis of γ-D-glutamyl-L-tryptophan, a known immunomodulatory peptide
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NF-κB inhibition subsequently impedes NLRP3 inflammasome activation, blocking its normal upregulation of proinflammatory cytokines, including IL-1β and IL-18 [88]
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