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Redox signaling in drug-tolerant persister cells as an emerging therapeutic target - ScienceDirect
Despite NS3h being well-studied, its contribution to HCV replication remains incompletely understood, and it is unclear which RNA structures are unwound by NS3h
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Indeed, G6PD overexpression suppressed Ang II-induced elevation of IL-1β and IL-6 protein levels, and TpNOX overexpression reversed this blockade (Fig
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