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As mentioned above, K1–SEMA and K3–SEMA interfaces are largely overlapped, meaning that once the SEMA of c-MET II is occupied by the K1 of HGF I with high affinity, the K3 of HGF II cannot bind to the same SEMA

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In particular, this may be substituting NO-effect [15] (and thereby, lesser NO-tissue values), additive and/or synergistic effect (NO fully implemented in the healing, and thereby, in relation to the NO-significance in the muscle and tendon healing [39,77], achieved myotendinous recovery) related to the own activity of BPC 157 administration in the rats having myotendinous lesions

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