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The therapy counteracted both early and delayed neural hippocampal damage, achieving full functional recovery (Morris water maze test, inclined beam-walking test, lateral push test) [38], as assessed at 24 h and 72 h of the reperfusion

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[8] This supports the involvement of ERK1/2 under the study conditions, but it does not prove that ERK1/2 is the only pathway involved or that it sits directly below FAK in a single BPC-157 signaling chain—the FAK and ERK findings came from different experimental systems